Max in WT synaptoneurosomes, suggesting that Src signaling may very well be downregulated in KI synapses. Then again, our power to rescue SERT functionality in KI midbrain synaptoneurosomes by the inhibition of FAK signifies elevated FAK signaling downstream of your Pro32Pro33 mutant, as confirmed by enhanced pFAK localization in five-HT https://dietrichz107frc9.ktwiki.com/user