Preliminary scientific tests indicate conolidine may possibly inhibit unique ion channels, decreasing neuronal excitability and restricting pain alerts. This mechanism is especially related in neuropathic soreness, in which abnormal signaling causes persistent irritation. Also, conolidine seems to affect G protein-coupled receptor (GPCR) pathways integral to ache notion. Computational... https://luthern790rep8.westexwiki.com/user