Preliminary studies indicate conolidine may possibly inhibit unique ion channels, decreasing neuronal excitability and restricting discomfort signals. This mechanism is particularly pertinent in neuropathic agony, wherever irregular signaling brings about persistent soreness. Moreover, conolidine appears to affect G protein-coupled receptor (GPCR) pathways integral to pain notion. Com... https://anciusa173xqj0.wikicommunications.com/user